Wednesday, May 22, 2019

Stress, Dieting, and Periodontal Disease

Christine Dursunian Principles of Dental Hygiene II Professor Willis Research Paper Stress, dieting and periodontic unhealthiness and to compromised systemic health. Stress, dieting, cultural diversity, foundation of health and periodontic disorder The average person would never think that all quartette of these topics would coincide with matchless an some other. They do, each affecting the other as stepping stones towards periodontal disease. periodontal disease, including periodontal disease and gingivitis, be chronic, bacterial transmittings and inflammatory diseases affecting the periodontium ( weaves that support the teeth). periodontic diseases argon the most prevalent chronic diseases affecting children, adolescents, adults, and the elderly. The periodontium is a complex, highly specialized, shock-absorbing and pressure-sensing system consisting of four interrelated threads supporting the teeth cementum, periodontal ligament, alveolar bone and junctional and sulcular epithelia (1). Periodontal disease can affect one or many teeth. It can withal lead to progressive bone loss around teeth, which can lead to loosening and possible loss of the teeth if left untreated.There are many factors to periodontal disease that have been proven to be directly related to this growing disease, in certain populations and cultures that have been proven to stand out more than others. through and through research and advanced studies with guidelines of evaluation, filtrate and dieting has been assignn to be associated with periodontal disease(2). These additional factors involve diet, lifestyle, cultures, also including collective types of strains in ones e preciseday life. Periodontal disease is an infection of the tissues that support the teeth.These infections are associated with specific pathogenic bacteria that colonize the subgingival area. When the teeth are being supported by the gingiva the gingiva does not bandage to the tooth firmly as one might thin k. Part of the tooths anatomy consists of a shallow v-shaped gaped called the sulcus which exists between the tooth and the gingiva. Periodontal disease affects this gap causing the tissue supporting the tooth to break down. Periodontal disease transitions through different levels to become what it is.Research studies indicate that a specific set of guidelines during evaluation of the association of stress and periodontal disease are important. When proposing experimental approaches, specifically in psychosocial stress and periodontal disease present studies and future experiments show the next six factors to be very(prenominal) useful. Periodontal disease is measured as unique disease outcome and should not be included in a composite index with other viva voce diseases(3). Validated instruments are assessed for stress, distress, and copying behaviors.These helpful instruments, validated by prior studies, also on the population for each applied for research. Indications of complia nce with viva voce health organization system should measure at-risk behaviors. Including oral health behaviors much(prenominal) as preventive dental visits, regulations of oral hygiene regimens, and an assessment of brass instrument, gingivitis, and other alive disease. By rigorously establishing psychosocial factors such as stress distress, and coping behaviors are true risk factors for periodontal disease case studies and case-control series generate hypotheses (4).These hypotheses are over large cross-sectional and longitudinal epidemiological studies. Studies in which mechanisms of psychosocial stress or distress show the application of periodontal disease The necessity to show affinity and correlation of applying different assessments such as biochemical mediators of stress, immune functions, or neurological and endocrine alterations as well as behavioral changes are significant in these cases(5). Lastly are randomized controlled trial methodology, the intervention of stu dies using stress management to establish efficacy of modification of stress as modality of stress-associated disease.Managing these controlled trials allows to reduce stress or distress. Periodontal disease has been associated for years with risk factors such as oral environment, age, female hormones, family factors, smoking and nicotine, and diseases associated with periodontal disease. When discussing oral environment the first issue that comes to mind is lack of oral hygiene. Lack of oral hygiene encourages bacterial buildup and plaque formation, which puts the oral cavity into a very susceptible position for periodontal disease.Inadequate contoured restoration has also created rise to periodontal disease for its restorations of fillings and crowns. By poor dentistry the restorations help trap for debris and plaque because of its defect in contour. Anatomical tooth abnormalities are influential risk factors just as poor restorations would be, because of these abnormalities the t eeth are not aligned in their born(p) state causing food particles to build up to the ladder of periodontal disease causing plaque and bacteria formation. Lastly would be the anatomy of the three molars, also known as the wisdom teeth.This tooth especially is a known to be a major breeding ground for bacteria, by reason of its mending and patients finding difficulty reaching posteriorly to the mouth while brushing. Other studies of this particular tooth show the unique tissues surrounding this molar region has been destructed of the plaque formation and the tissue becoming more sensitive when the third molar is meeted, meaning when the tooth is wedged between another tooth and the jaw. Next commonly associated risk factor with periodontal disease is age.Another problem in aging, comes the increase of obtaining periodontal disease. Studies have shown that thirty percent of the adults in America have periodontal disease and mostly found in people over the age of seventy years old, eighty-six percent have periodontitis (6). Rare cases it has been shown for young adolescents who are subjected to this disease. Adding to this is family factors, periodontal disease a great deal occurs to members of the same family. A mixture of factors belong to this title, such as intimacy, genetics and hygiene.A considerably important factor is smoking and nicotine. Being the most significant factor, causing bone loss and gingival recession and inflammation by reducing the amount of oxygen present in the gingiva tissue. When nicotine combines with oral bacteria, the production levels expectations are greater. Lastly are diseases associated with periodontal disease, such as diabetes, type one and two, osteoporosis, herpes related gingivitis, human immunodeficiency virus associated with gingivitis, autoimmune disease (7).Others diseases of genetic disoblige are also at high risk for periodontitis. Contemporary conceptualization of the stress process supports the evaluation of s tress at three levels. Stressors, moderating and mediating factors, and stress reactions. These three factors have emphasized the process and the unity stress can have on periodontal disease, including emotions and coping(8). These stress responses would be determined primarily by the process that makes personalized perception of a stress indicator or a threat to ones health.The aim of each study is to unfold influential factors such as personality traits, coping strategies, and experimentation of referenced information (9). The resolution of all studies suggested that depression, stress, and salivary cortisol are important correlates of periodontal disease. Therefore, it is likely that periodontitis is related to immunologic and behavioral changes related to psychologic states. Salivary cortisol seemed to have different associations with periodontal disease, because of the outcomes in models involving stress compared to models involved with depression.Periodontitis is indicated whe n addressing depression or stress. By strongly suggesting that stress, distress, and inadequate ways of handling difficult situations of coping are important risk indicators for periodontal disease. Furthermore, it is likely that systemic disease associated with periodontal disease such as diabetes, cardiovascular disease, preterm delivery and osteoporosis may share psychosocial stress as a common risk factor.During these early beginnings of study and research, requirement for one to fully understand the molecular and cellular basis of the role of stress, and in turn these studies may lead to effective intervention strategies that minimize or deviate stress as a contributor to periodontal disease. Research has also proven within certain limitations this systematic review showed a positive relationship between stress and psychosocial factors and periodontal disease.However, caution should be used when interpreting this review because the different methodologies used in the included studies may have an impact on the results of the reports. The difficulties inherent is isolating the variable of stress, the lack of a reliable standardize psychological analysis to quantify and define most psychiatric disturbances, the individualist ability of patients to cope with negative life events, and the different types and clinical parameters used to determine periodontal tissue breakdown may cat as a confounding biases and cause result distortion at several stages. lthough a positive relationship was observed between stress and periodontal disease, further exercise research is need to determine the impact of stress and psychological factors as risk factors for periodontal disease. Reference Ng SKS, Leung WK. A community study on the relationship between stress, coping, affective dispositions and periodontal attachment loss. Community Dental Oral Epidemiol 2006 24 252-66 Page RC, Kornman KS. The pathogenesis of human periodontitis an introduction. Periodontol 2000 1997 14 9-11 Robert J. Genco, Alex W. Ho, Jeffrey Kopman, Sara G. Grossel, Robert G.Dunford and Lisa A. Tedesco. Models to Evaluate the quality of Stress in Periodontal Disease. Department of Oral Biology, Periodontal Disease Research Center, School of Dental Medicine, State University of New York at Buffalo. Vol. 3, No 1, 288-302 July 1998. Amy E. Rosania, Kathryn G. Low, Cherly M. McCormick, and David A. Rosania. Stress, Depression, Cortisol, and Periodontal Disease. Department of Psychology, Bates College, Lewiston, ME. record 80, Number 2260-266 February 2009. Breivik T, Thrane PS, Murison R, Gijermo P. Emotional stress effects on immunity, gingivitis, and periodontitis.Eur J Oral Sci 1996 104327-334 Green LW, Tyron WW, Marks B, Juryun J. Periodontal disease as a function of life-events stress. J Human Stress 19861232-6 Annsofi Johannsen, Gunnar Rylander, Birgitta Soder, and Marie Asberg. Dental Plaque, Gingival Inflammation, and Elevated Levels of Interleukin-6 and Cortisol in Gingi val Crevicular Fluid From Women with stress-related Depression and Exhaustion. Volume 77 November 8 2006 771403-1409 Genco RJ, Ho AW, Grossi SG, Dunford RG, Tedesco LA. Relationship of stress distress and inadequate coping behaviors to periodontal disease.J Periodontol 199970711-23 Daiane C. Peruzzo, Bruno B. Benatti, Glaucia M. B. Ambrosano, Getullo R. Nogueria-Fiho, Enilson A. Sallum, Marcio Z. Casati, and Franciso H. Nociti Jr. A Systematic round off of Stress and Psychological Factors as Possible Risk Factors for Periodontal Disease. Volume 78- Number 8. 2007781491-1504 Kaufman E, Lamster IB. Analysis of saliva for periodontal diagnosis- A review. J Clin Periodontal 2000 27 453-465 Arowojolu MO, Onyeaso CO, Dosumu EB, Idaboh GK. Effect on academic stress on periodontal health 2006299-13

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